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Overactive ERBB4 protein in excitatory neurons drives Alzheimer's pathology
A study published in Nature found that aberrant activity of the ERBB4 protein in excitatory neurons promotes the hallmark pathological changes of Alzheimer's disease. The research also suggests that neuroinflammation may not be necessary for early synapse loss, challenging a widely held assumption about how the disease progresses. The findings could point to new therapeutic targets for Alzheimer's treatment.
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